Bruising brain swelling
Spinal Brain Injury honey.
Spinal cord injury - optionspinal cord injury, characterized by the appearance in the spinal cord of reversible (functional), as well as irreversible (organic) changes in the form of foci of necrosis, crushing, hemorrhages, leading to partial damage or to a morphological break of the spinal cord.
• Pathomorphological changes can be both primary - in the place of application of traumatic force, and secondary - due to circulatory and circulatory disorders of CSF.
• Morphological spinal cord breakto be anatomical (with a divergence of ends) and axonal, when the integrity of the spinal cord is externally preserved, although its conductor systems at the level of trauma are destroyed.
• A complete anatomical spinal cord break occurs rarely.
• The contusion of the spinal cord is not classified according to the degrees of severity. Depending on the depth of the lesion, a neurological deficit of varying severity is observed.
• Phase flow. A spinal cord injury is accompanied by a spinal shock (see Spinal-Spinal Injury Trauma), which masks the true picture of spinal cord injury. Only after the resolution of shock there is a persistent symptomatology, reflecting the degree of damage to the spinal cord.
• Conduction disruption syndrome is the most common manifestation of a spinal cord injury.
• Complete conduction disorder syndrome
• Violation of motor functions in the form of flaccid paralysis, areflexia
• Disorders of all kinds of sensitivity according to the conductor type (its absence below the level of damage)
• Vegetative symptoms: disorders of thermoregulation, trophic (dry skin, rapid formation of decubitus)
• Severe dysfunction of the pelvic organs.
• Partial conduction disorder syndromeis characterized by the same disorders as when it is completely disturbed, but at the same time they reveal signs that indicate partial preservation of the conductivity of the spinal cord (detection of movement disorders or sensitivity below the level of damage, expressed to some extent).
• Complete transverse spinal cord injury(morphological break) - a syndrome of complete conduction disruption in the acute period and automaticity of the distal part of the brain below the level of interruption in later periods. Prolonged priapism and early trophic disorders indicate irreversible brain damage.
• For a neurological examination, it is first necessary to find out whether there is a picture of complete transverse damage to the spinal cord or only a partial loss of its functions
• CSF - an admixture of blood, with liquorodynamic tests sometimes reveal a different degree of blockade of the subarachnoid space
• Radiography of the spine in two projections, myelography
• CT and MRI.
Treatment is conservative - see Trauma to the spinal cord.
• Ascending edema of the medulla oblongata
• Visceral: the emergence of an ascending urinary tract infection, urogenital sepsis, bronchopneumonia
• Education of pressure sores
• Articular contractures
• Deep vein thrombosis, thromboembolism of various locations. Forecast
• Relatively unfavorable. You can not expect a significant improvement in the condition, since regeneration with an inaccurate comparison of the axon ends is negligible
• If the clinical picture is completetransverse lesion in the next 48 hours, there are no slight signs of restoration of functions, this indicates an organic, and not functional, nature of the lesion, which is considered a poor prognostic sign (irretrievable loss of function)
• Spinal roots, being peripheralneurons, are capable of regeneration, but only in cases when the whole outer shell of the root remains at least. After a complete rupture, the regenerating axons of the central stump are separated from the distal stump, so there is no coalescence.
Synonyms. Spinal cord contusion
See also Hematomyelia. The compression of the spinal cord. Injury of vertebral-spinal
• S14.0 Concussion and edema of the cervical spinal cord
• S24.0 Bruising and edema of the thoracic spinal cord
• S34.1 Other trauma of lumbar spinal cord
Handbook of Diseases. 2012.