Without signs of perifocal edema
Hello! I'm 30 years old and I'm worried about frequent headaches. Pain in the neck and dizziness. Please comment the results of my study on MRI: the lateral ventricles of the brain are not dilated, asymmetric, with a moderate zone of peripheral gliosis. The third ventricle has a width of 0.4 cm. The 4th ventricle is unchanged. Basal tanks are not changed. Chiasmatic region without features. The pituitary gland is located intrasellarly, of ordinary size. The pituitary gland has a normal signal. Subarachnoidal convective space is locally unevenly minimally widened, mainly in the region of the frontal and parietal lobes. The middle structures are not displaced. Tonsils of the cerebellum are located at the level of BZO. In the white substance of the frontal and parietal lobes, multiple small foci of gliosis are defined, with dimensions from 0.2 to 0.4 cm without signs of perifocal reaction. CWO-free features. The signal from the mucous membrane of the nasal cavity, maxillary sinuses and cells of the latticed labyrinth due to its edema is amplified. Conclusion: MR picture of minimal expressed arachnoidal changes of the cerebrospinal fluid. Focal changes in the substance of the dystrophic yostga.
You do not need a neurosurgeon!
Question No. 2382. Date of reply 13.07.2009
Hello dear doctors! A visit to the doctor after the MRI of the brain is postponed due to his well-deserved summer vacation, but the conclusion of the doctor who conducted the MRI scan is somewhat frightening and naturally I want to know how serious the situation is. So: I'm 35 years old, three years old (according to the appointment of a cardiologist) I take medications that reduce pressure, headaches are not intense, but frequent. Sometimes there is numbness in the fingers, and nocturnal leg cramps. In addition to cholesterol in the borderline, the remaining blood and urine tests are normal. EEG showed signs of intracranial hypertension. Work on 90% is connected with the computer. description of the MRI study: On a series of MRIs weighted by T1 and T2 in three projections, sub- and supratentorial structures are visualized. In white matter, the right frontal and left parietal lobes define single small foci of demyelination of 0.3 cm and 0.4 cm, respectively, without signs of perifocal edema. Lateral and III-th ventricles of the brain of the usual configuration and size. IV th ventricle, basal cisterns moderately expanded. Chiasmatic region without features. The pituitary body is 1x0.5x1.5 cm in size, its tissue has a normal signal. The pineal gland is 0.9x0.6 cm in size. Its structure shows cystic formation with fluid signal characteristics. Subarachnoidal convection spaces and furrows are locally unevenly expanded, mainly in the frontal-parietal lobes. The median structures are not biased. Tonsils of the cerebellum are usually located. The town is small and the choice of specialists, alas, is not great. In advance I thank for consultation.
Judging by the description of tomograms, operating on youthere is nothing, and, in fact, a neurosurgeon you do not need. Address to the neurologist if the concrete doctor does not arrange, find another, neurologists-not such rare or infrequent experts.
Question No. 2342. Date of reply 18.06.2009
Hello, my father (1964g.) Three years ago there was a trauma to the skull (a gunshot wound wound, localization to the occipital region), as a result of which trepanation of the skull was performed, bone was removed in the occipital region of approximately 5.5 cm. Recently, CT scan of the brain, and here is the conclusion: in the occipital bone, trephine defect of the vault 55mm. In the right occipital lobe, the uneven decrease in the density of brain matter is 10-14 units. HU, with relatively clear contours. Ventricular of the usual form, lateral ventricles symmetric, moderately narrowed, in periventricular departments the density of white in-va brain 37 ed. H. sylvic gaps are not enlarged. The secondary education is not displaced. Subarachnoid spaces are unevenly widened, with single cystlike formations in the frontal-parietal areas, convective furrows are visualized satisfactorily. Conclusion: CT-signs of the state after trepanation of the skull, cicatrical-atrophic changes in the right occipital region, intracranial hypertension, arachnoidal changes in the liquorcystic nature. After trepanation, his vision deteriorated, the trephination defect in the plate was not closed, when the A / D rises, the vessels bulge and a strong pulsation is observed and headaches begin. What do you advise to do? What services can your clinic offer and how much does it cost? Do I need to delete the cysts? Is it possible to restore vision?
Plasticity of skull defect is possible, but you mustto understand that this operation is performed mainly for the purpose of protecting the brain located under the defect area. She does not have a relationship to restore vision. And the prospects for vision in your case are likely more than modest.
When carrying out a computer tomography of the headthe father was established: In the temporal lobe of the right hemisphere, a pathological formation d 2.6 x 1.9 cm of increased density to 64 units is determined. heterogeneous structure and pronounced perifocal edema. In the parietal lobe of the left hemisphere there is a rounded formation d 2.1 cm from the SC 45 E.H. heterogeneous structure and moderately pronounced perifocal edema. The median structures are asymmetric, displaced to the left by 0.4 cm. The right lateral ventricle is compressed in the region of the body and the anterior horn. External liquor spaces are smoothed over the right hemisphere of the brain. Conclusion: CT signs of volumetric formations of both hemispheres of the large brain with mass-effect to the left and compression of the right lateral ventricle (more likely mts)
What's the question? in the conclusion everything is written. Showing MRI of the brain with contrast enhancement and further follow-up and treatment.
Question No. 2033. Date of reply 04.02.2009
Hello! To me 35 years from 14 years to me have diagnosed an epileptic syndrome have made MRT. On a series of MR Tomograms of external pT1 and T2 in three projections are visualized sub and subbrantental structures. Changes of focal and diffuse character in a brain substance are not revealed. The ventricles of a brain of the usual sizes and configurations. 3rd and 4th ventricles basal cisterns are moderately dilated. Chiasmatic region without features. Pituitary tissue has a regular signal. Subarachnoidal convective spaces are locally unevenly expanded. Primarily in the frontal theme -s' shares. The median structures are not biased. Tonsils of the cerebellum are usually located. The mucous membrane of the maxillary sinuses is thickened due to moderate edema. In the right upper maxillary sinus, a 2.0-by-1.8 cm cyst is visualized. At the border of the scanning field in the region of the nasal septum, a rounded form is defined with clear, even outlines hyperintensive on T1 VI and iso-intensive on T2 of VI in the size of 0.9 by 0.9 cm-lipoma and is questionable. On the series of MR angiograms. performed in the TOF mode. In the axial projection, internal sleepers are visualized. the main.intracrinal segments of the vertebral arteries and their branching. Variant of development of the Willis circle in the form of absence of blood flow along the right posterior connective artery. It determines the lumidity and irregularity of the blood flow along the intracranial section of the PA. The lumens of the other blood vessels are uniform. The walls are symmetrical. sites with pathological blood flow were not detected. Conclusion MR card dilatation of external cerebrospinal spaces. Rhinosinusopathy. It is recommended to consult an ENT doctor. Variant of development of the Willis circle. The crippling left PA. Can such a diagnosis give epileptic seizures? Sincerely, Julia.
Judging by the description, there are no morphological reasons for convulsive seizures. But this does not mean that there are no other reasons. Focus on EEG mapping.
Question No. 1768. Date of reply 06.10.2008
The dear doctor! My son Vladimir is 22 years old. At the age of 14, the son was found an arachnoid cyst of the fronto-temporal parietal region to the left. Hypertensive-hydrocephalic syndrome. Dimensions cyst 73x74x44 mm displacement of the median structures of the brain to 5 mm to the right. From the age of 15, diabetes insipidus, an average degree of severity, which is not amenable to treatment, due to persistent headaches, worsening with the administration of medications prescribed by an endocrinologist. In January 2008, the son was beaten by a group of teenagers, examined by a therapist, a neurologist, an echo of the EE, an X-ray of the skull made, and no deterioration was found. He was treated for concussion of the brain, he was treated for a month. In August 2008, there were bursting headaches, the temperature rose, there was no vomiting. Have made MTR - the conclusion. chronic subdural hematoma of the left hemisphere of the brain, signs of lateral dislocation. According to the description of MTP in the frontotemporal temporal region, a convectional subdural hematoma with a high-efficiency signal in T2 and an iso-hypertensive T1 signal of WI, up to 2.7 cm in width, up to 14.7 cm in length, Hematoma is compressed into the adjacent parts of the brain. The middle structures are shifted to 0.7 cm to the right. Since August 23, 2008, the patient is in the neurosurgical vascular compartment of the OKB of Ivanovo. September 1, 2008. Tomography of the brain was made. an extensive subdural hygroma of the left frontal-frontal lobe with the expansion into the Sylvian gap V = 150 ml with a displacement of the middle structures up to 3 mm. On the tomogram, the cysts were not visible. September 3, 2008 The patient underwent surgery. Milling trepanation of the skull in the projection of the middle point of Cronlein on the left with the removal of the chronic subdural hematoma. The operation did not bring relief. September 15, 2008 The arachnoid cyst of the left lateral cleft. Chronic subdural hematoma of the left frontal-parietal region. Tomography without positive dynamics. The patient regularly took from 14 years a diuretic - diacarb and vascular drugs. After the operation, capitally, Cavinton, mexidol, and euphiline. Patient 3 group disability 1 degree of restriction. But after the operation, the headaches of the bursting nature began to disturb more strongly, the patient does not sleep well at night. All prescribed medications (diuretic and vascular) take in tablets. Sincerely, Smirnov family, Ivanovo
Hello. Send the latest pictures and sign up for a consultation. The situation is serious. It is possible that it may be a question of a second operation.
Question No. 1732. Date of reply 11.09.2008
Hello! I'm 33 years old. I have the following question. The left part of the head hurts (an apex, a temporal part), sometimes weak, sometimes moderate pains. Also complaints of dry cough (periodically), there is an increase in veins on the left hand, a weak numbness of the left arm, fatigue at the end of the day. wake up in the middle of the night. Appetite normal, weight constant 68 kg. Earlier I had recurrent secondary pulmonary tuberculosis in 2007-2008. During the treatment of tuberculosis there was a long insomnia, somewhere 10 months, now the sleep more or less normalized. chronic chronic runny nose, sinusitis. seasonal allergy. On 30.01.2008 an MRI was performed at the Medical Center "Origo", Astrakhan. Survey report: Median structures are not displaced. In the parenchyma of the brain, the pathological intensity of the signal is not detected. Hippocamps are symmetrical. Ventricles of usual shape: lateral asymmetric, their width at the level of the body. on the right-11 mm. left 15 mm. A moderate expansion of the cortical sulcus and subarachnoid space of the convectional parts of the brain, intercostal cistern and cisterns of the posterior cranial fossa is determined. The pineal region, the pituitary gland is not changed. The structures of the posterior cranial fossa, the visible sections of the cranial nerves, the craniospinal transition without singularity. There is a parietal edema and hyperplasia of the mucosa of the lower parts of the frontal sinus and the cells of the latticed labyrinth. There is a thickening of the scales of the frontal bone. The upper sagittal sinus and superficial ascending veins of the fronto-parietal region are somewhat hypertrophied. Conclusion: There were no focal signals in the brain. Phenomena of mild, open, mainly external hydrocephalus. Signs of frontoethmoiditis. The analysis of blood and urine is normal. Also I want to add that I have been working on the computer all the time since 2000. At the moment I am treated with mummies, herbs, nut butter, I constantly drink the healing and table mineral water "Brakshun" (based on rock oil). What can you advise in this situation. Sincerely, Serik
Hello! You need to be seen at the ENT doctor.
Question No. 1705. Date of reply 14.08.2008
Hello, I'm 23 years old, on a series of receivedMR-tomograms in the sagittal, axial and coronary projections in T-1 and E-2 regimens in the basal sections of the right temporal lobe focus high in the T-2 mode of the MR signal with dimensions of 34x16x26 mm with clear contours. The median structures are not biased. Ventricular system of usual configuration. Pituitary body of usual size and structure. The craniovertebral transition is not changed. Conclusion: MR-signs of the arachnoid cyst of the right temporal lobe. When examining the fundus, it is established that the DZN is hyperemic, the borders are slightly stilted under the vessels and on the nasal side (more in the OS); veins enlarged, strained (OS greater than OD); in OS - easy exudation in a vessel. funnel. Initial stagnant DZN of both eyes (more pronounced in the left eye). Tell me, is it dangerous, is surgery necessary and what is better to do in this case?
Question No. 1689. Date of reply 05.08.2008
Hello! Very much I hope for your help, advise us, please, what our further actions and methods of treatment of illness of my aunt (age of 56 years) should be. The data of M RT - on the received MR-tomograms convective, parasagittally in the left parietal lobe there is a rounded tumor formation with precise uneven contours, a non-uniformly hyperintensive signal on T2VI and hypointense on T1VI, intensively heterogeneously accumulating contrast medium. surrounded by a vast perifocal edema, a broad base adjacent to the inner closure plate of the cranial vault, separated from the brain tissue by the liquor layer, with dimensions 36x30x26 mm. Identified single foci of a hyperintense signal located in the subcortical white matter of the frontal and parietal lobes from two sides. Focal changes of the trunk, cerebellum not revealed. The ventricles of the brain are not dilated, the triangle of the left lateral ventricle is compressed, the anterior horns of the lateral ventricles are surrounded by a hyperintense signal at T2 VH. The median structures are not biased. Subarachnoid spaces of the cerebral hemispheres and cerebellum are not changed. Cellar region and cerebrospinal transition without visible changes. On a series of MRA mode 3 DTOF, the topography of the vessels of the Willis circle is preserved, the intensity of the MR signal of the main arteries of the brain is not changed, the sites of pathological constriction and the expansion of cerebral vessels are not revealed. There is markedly hypertrophic middle shell artery to the left, feeding the branch of tumor formation. Convincing signs of an effect on the sagittal sinus on the part of education have not been revealed. Pneumotization of the paranasal sinuses is retained, the increase in the MR signal from the cells of the mastoid process and the pyramid of the temporal bone on the right are determined, signs of vestigial changes in the middle ear. The multiple rounded formations in the hypodermic fatty tissue of the head with a homointense signal at T2 and T1VI diameter from 8 mm to 20 mm are visualized, the largest formation has a nonuniform MR structure. Conclusion: The MP pattern of the formation of the brain in the indicated location (most likely the convective parasagittal meningioma of the left hemisphere of the brain). Focal changes in the substance of the brain of vascular genesis. Sincerely, Svetlana.
Svetlana, for the beginning with pictures on reception to the neurosurgeon. [email protected]
Question No. 1587. Date of reply 30.05.2008
To the son of 8 years by results of KT have put the conclusionretrocerebellar cyst. According to the survey: The median structures of the brain are not displaced. The hollow system is not expanded - the width of the ventricle is 2.2 mm. Behind the worm of the cerebellum is the formation of a reduced (liquor) density of 11.6 mm in diameter with a rather distinct contour without signs of perifocal edema and mass effect. Other foci and formations of pathological density and edema are not determined. On how much it is serious and what will be the treatment (or surgery is needed)?
Hello, our clinic provides neurosurgical care only to patients from 15 years of age. But you can send pictures to the site and you will be answered.
E-mail: [email protected] Phones: 252-35-56, 945-80-22